Calcium Signalling and Disease - Molecular Pathology of Calcium - Subcellular Biochemistry Vol 45

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Cell Biol.

Metabolic signaling functions of ER–mitochondria contact sites: role in metabolic diseases

Kayed R. Shankar G. Demuro A. Giuffrida M. Xia W. Kuo Y.


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  • Store-Operated Calcium Channels | Physiological Reviews!
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Gong Y. Haes A. Meyer-Luehmann M. Deshpande A. Glabe C. Goldman W. Lansbury P. Teplow D. Rovira C. Verdurand M. Aging , in press.

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Google Scholar. Buckingham S. Geerts H. Terry A. Kadir A. Bartus R. Wevers A. Wang H. Garashchuk O.

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Parameshwaran K. Kelly B. Carette B. Snyder E. Dewachter I. Aging 30 , — Wang Y. Fox A. MacManus A.

Nimmrich V. Decout A. Acta , — Terzi E. Avdulov N. McLaurin J. Cribbs D.

Calcium and Calmodulin

Kremer J. Mingeot-Leclercq M.


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Lipids , 57 — Sokolov Y. Capone R. Arispe N. Lin H. Quist A. Pollard H. Lashuel H. Durell S. Jang H. Inoue S.

Store-Operated Calcium Channels

Diaz J. LaFerla F. Shtifman A. Aging doi: CrossRef Google Scholar. Schapansky J. Casley C. Shaked G. Cellular calcium homeostasis, and thus calcium signalling, is mainly regulated by membrane intrinsic proteins and calcium sensor proteins. Both classes may be involved in pathological processes that affect both human and animals, ranging from common and important diseases e. Clearly, the topic has now become not only very large, but also very stimulating.

Calcium-independent phospholipases A2 and their roles in biological processes and diseases

Its extensive critical coverage is likely to eventually stimulate fruitful interdisciplinary discussions. JavaScript is currently disabled, this site works much better if you enable JavaScript in your browser. Biomedical Sciences Human Genetics. Subcellular Biochemistry Free Preview. Represents the first book on this emerging and important topic Sets forth an interdisciplinary approach, from biochemistry to clinical medicine Provides new insights into the underlying causes of congenital and acquired diseases Points the way to future, effective therapies see more benefits.

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